Cranial Nerves Lab Evaluation at Cape Fear Community College

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Free Cranial Nerves Lab Evaluation at Cape Fear Community College Questions

1.

Frontal lobe lesions or cribriform or ethmoid bone trauma can potentially cause damage to which of the following cranial nerves?

  • Olfactory

  • Optic

  • Oculomotor

  • Trochlear

  • Trigeminal

Explanation

A. Olfactory The olfactory nerve (Cranial Nerve I) passes through the cribriform plate of the ethmoid bone. Trauma to this area—such as head injury impacting the frontal lobe or fractures through the cribriform plate—can shear the olfactory nerve fibers, resulting in loss of smell (anosmia). This nerve is highly vulnerable in anterior skull base fractures and frontal head trauma due to its direct connection to the frontal lobe and its passage through delicate bony foramina.
2.

When testing the vagus nerve, the nurse should assess for:

  • Facial asymmetry.

  • Tongue movement.

  • Hoarseness.

  • Proprioception.

Explanation

The vagus nerve (CN X) innervates the larynx and pharynx. A nurse assesses it by checking swallowing, gag reflex, and voice quality. Hoarseness indicates possible dysfunction because CN X controls vocal cord movement. Damage may result in a weak, hoarse, or absent voice, which is a key finding when evaluating vagus nerve function.

3.

Cranial nerve one (CN I), the _________, is instrumental in our sense of _________.

  • vestibulocochlear nerve / balance

  • trigeminal nerve / facial sensation

  • olfactory nerve / smell

  • optic nerve / vision

Explanation

Cranial Nerve I is called the olfactory nerve and its primary function is to carry sensory information related to smell from the nose to the brain. This nerve is responsible for detecting odors and transmitting smell signals to the brain for interpretation.

4.

The glossopharyngeal nerve is assessed by asking the client to __________ and move their __________ __________.

  • smile / eyebrows / upward

  • swallow / soft palate / up and down

  • protrude the tongue / tongue tip / side to side

  • clench teeth / jaw / forward and backward

Explanation

The glossopharyngeal nerve (CN IX) is evaluated by observing swallowing and soft-palate movement. Asking the client to swallow and watching the soft palate move up and down assesses gag reflex and pharyngeal muscle function. Proper elevation indicates intact CN IX function, essential for swallowing and airway protection.

5.

Cranial Nerve 6 innervates which of the following? Select all that apply.

  • Lateral rectus

  • Superior rectus

  • Medial rectus

  • Inferior rectus

  • Superior oblique

  • Inferior oblique

Explanation

A. Lateral rectus Cranial Nerve VI (Abducens nerve) innervates the lateral rectus muscle. This muscle abducts the eye, moving it outward away from the nose. When CN VI is damaged, the affected eye may drift inward (medial deviation), and the patient may experience horizontal double vision that worsens when looking toward the affected side.
6.

How does the nurse test cranial nerve two (CN II)?

  • Ask the client to identify different tastes.

  • Ask the client to clench their teeth.

  • Ask the client to cover each eye separately and read the Snellen chart.

  • Ask the client to shrug shoulders against resistance.

Explanation

Cranial nerve II is the optic nerve, responsible for vision. The nurse tests CN II by assessing visual acuity, most commonly using a Snellen eye chart. Each eye is tested separately to identify deficits in central vision. This helps determine clarity of sight and identifies potential neurological or ocular problems such as optic neuritis or visual field loss.

7.

Ketamine administration can result in nystagmus. Which of the following cranial nerves is responsible for this phenomena?

  • Oculomotor

  • Cranial nerve 6

  • CN IV

Explanation

B. Cranial nerve 6 Cranial nerve VI (Abducens nerve) innervates the lateral rectus muscle, which abducts the eye. When ketamine is administered, it can affect ocular muscle control and cause nystagmus, including lateral gaze nystagmus. Because CN VI controls side-to-side eye movement, dysfunction or overstimulation of this pathway can contribute to the horizontal nystagmus often seen with ketamine use.
8.

Cranial nerve three (CN III) allows movement of the eye muscles and constriction of the pupils.

  • True

  • False

  • Controls hearing and balance

  • Controls swallowing and gag reflex

Explanation

Cranial nerve III (Oculomotor nerve) is responsible for most extraocular eye movements (upward, downward, and inward gaze), raising the eyelids, and pupil constriction via parasympathetic fibers. It plays a key role in eye tracking, accommodation, and response to light. Dysfunction can lead to ptosis, double vision, and a dilated pupil that does not constrict.

9.

Click one area on the image to show where the nurse would assess cranial nerve 11. Select your answer by clicking the desired location on the image below. To move a pin, click another location on the image. To remove a pin, click it once.

  • Side of the neck over the sternocleidomastoid muscle

  • Upper shoulder over the trapezius muscle

  • Forehead center

  • Tip of the chin

  • Tongue surface

Explanation

Correct Selections Explanation A. Side of the neck over the SCM Cranial Nerve XI innervates the sternocleidomastoid muscle. The nurse palpates the SCM by asking the patient to rotate the head against resistance. This assesses neck strength and CN XI function. B. Upper shoulder over the trapezius The trapezius muscle is also controlled by Cranial Nerve XI. The nurse tests this by placing hands on the shoulders and asking the patient to shrug upward against resistance. Weakness suggests accessory nerve damage.

10.

Papillaedema, loss of vision, or a blurred optic disc are pathological findings of which of the following cranial nerves?

  • Olfactory

  • Optic

  • Oculomotor

  • Trochlear

  • Trigeminal

Explanation

B. Optic The optic nerve (Cranial Nerve II) carries visual information from the retina to the brain. Papilledema, blurred optic disc, and loss of vision are hallmark findings of optic nerve involvement. These findings typically indicate increased intracranial pressure or direct damage to the optic nerve. Changes at the optic disc are seen during fundoscopic exam and signal pathology affecting visual transmission.

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